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We show that CGNs isolated from Rag1 KO mice showed altered Ca2+ dynamics in the form of increased cytoplasmic Ca2+ activity, resulting in an enhanced Ca2+ efflux through ryanodine receptors. To investigate mechanisms underlying the loss of GABAergic inhibition in this model, we studied intracellular Ca2+ dynamics in cerebellar granule neurons (CGNs) isolated from Rag1 KO and wild-type (WT) mice. A loss of GABAergic inhibition has been described in the autism mouse model, the Rag1 knockout (KO). There is therefore a need for an improved method of automatically creating printouts of a computer display.Altered cytosolic Ca2+ dynamics in cultured cerebellar granule neurons from a genetic mouse model of autism.Īutism spectrum disorders (ASD) are a group of neurodevelopmental disorders characterized by social and communication deficits, and repetitive and restricted interests and behaviours. This of course has the disadvantage that a user of the computer display needs to understand the software which is required to create the printout.

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